Publications

– Pirola JP, DeForest P, Protachevicz PR, Fontenas L, Ferreira R, Pena RFO. Astrocytic Signatures in Neuronal Activity: A Machine Learning-Based Identification Approach. Cognitive Neurodynamics, 2025. DOI: 10.1007/s11571-025-10276-4.

– Fontenas L. Glial Plasticity At Nervous System Transition Zones. Biology Open, 2023. DOI:10.1242/bio.060037.

– Fontenas L and Kucenas S. Spinal cord precursors utilize neural crest cell mechanisms to generate hybrid peripheral myelinating glia. eLife, 2021. DOI:10.7554/eLife.64267. 

-Ali M, Latimer A, Wang Y, Hogenmiller L, Fontenas L, Isabella AJ, Moens C, Yu G, Kucenas S. Met is required for oligodendrocyte progenitor cell migration in Danio rerio. G3-Genes, Genomes, Genetics 2021. DOI:10.1093/g3journal/jkab265.

–Fontenas L, Welsh TG, Piller M, Coughenour P, Gandhi AV, Prober DA, Kucenas S. The neuromodulator adenosine regulates oligodendrocyte migration at motor exit point transition zones.Cell Reports, 2019. DOI:10.1016/j.celrep.2019.03.013.

-Mikdache A, Fontenas L, Albadri S, Revenu C, Loisel-Duwattez J, Lesport E, Degerny C, Del Bene F, Tawk M. Elmo1 function, linked to Rac1 activity, regulates peripheral neuronal numbers and myelination in zebrafish. Cellular and Molecular Life Sciences, 2019. DOI:10.1007/s00018-019-03167-5. 

– Fontenas L and Kucenas S. Motor Exit Point (MEP) glia: Novel Myelinating Glia That Bridge CNS and PNS Myelin. Frontiers in Cellular Neuroscience, 2018. DOI:10.3389/fncel.2018.00333. 

– Fontenas L and Kucenas S. Livin’ On The Edge: glia shape nervous system transition zones. Current Opinion in Neurobiology, 2017. DOI:10.1016/j.conb.2017.09.008.

– Fontenas L and Kucenas S. Glial Cell Development. Reference Module in Life Sciences, 2017 (book chapter). DOI:10.1016/B978-0-12-809633-8.12149-1.

– Fontenas L, De Santis F, Di Donato V, Degerny C, Chambraud B, Del Bene F, Tawk M. Neuronal ndrg4 Is Essential for Nodes of Ranvier Organization in Zebrafish. PLoS Genetics, 2016. DOI:10.1371/journal.pgen.1006459.

– Laquerriere A, Maluenda J, Camus A, Fontenas L, et al. Mutations in CNTNAP1 and ADCY6 are responsible for severe arthrogryposis multiplex congenita with axoglial defects. Human Molecular Genetics, 2014. DOI:10.1093/hmg/ddt618.